辐射诱导肺纤维化作为进行性纤维化模型：DNA损伤、炎症反应和细胞衰老基因的贡献

资源简介

辐射诱导肺纤维化作为进行性纤维化模型：DNA损伤、炎症反应和细胞衰老基因的贡献

【数据集背景】

本数据集源站标题为《Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genes》。配套论文《Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genes》发表于 Experimental Lung Research（2017），DOI：10.1080/01902148.2017.1318975。源站首次发布：2017-05-23。

【数据内容】

源站原始描述：Purpose/Aim of Study: Studies of pulmonary fibrosis (PF) have resulted in DNA damage, inflammatory response, and cellular senescence being widely hypothesized to play a role in the progression of the disease. Utilizing these aforementioned terms, genomics databases were interrogated along with the term, “pulmonary fibrosis,” to identify genes common among all 4 search terms. Findings were compared to data derived from a model of radiation-induced progressive pulmonary fibrosis (RIPF) to verify t…

【数据结构与技术规格】

文件数 6 个，合计 4.82 MB。ielu_a_1318975_sm7765.tif：image/tiff，3.49 MB。ielu_a_1318975_sm7764.tif：image/tiff，299.29 KB。ielu_a_1318975_sm7763.pdf：application/pdf，318.38 KB。数据集 DOI：10.6084/m9.figshare.5033381.v1。

【主题与分类】

源站学科分类：Medicine、Microbiology、Genetics、Molecular Biology、Ecology、Immunology。主题标签：pulmonary fibrosis、radiation、RNA sequencing、IPF。

【适用方向】

源站学科分类标注为 Medicine、Microbiology、Genetics、Molecular Biology、Ecology、Immunology。

【获取与许可】

源站页面：https://tandf.figshare.com/articles/dataset/Radiation_induced_pulmonary_fibrosis_as_a_model_of_progressive_fibrosis_Contributions_of_DNA_damage_inflammatory_response_and_cellular_senescence_genes/5033381

使用许可：CC BY 4.0（https://creativecommons.org/licenses/by/4.0/）。

规范引用：Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genes. Taylor & Francis. Beach, Tyler A.; Johnston, Carl J.; Groves, Angela M.; P. Williams, Jacqueline; N. Finkelstein, Jacob

【使用注意】

· 源站最后修改于 2023-06-06，引用时请注意版本时效。