尤文肉瘤与间充质干细胞差异表达的miRNAs：低表达miR-31对增殖与侵袭的影响

资源简介

尤文肉瘤与间充质干细胞差异表达的miRNAs：低表达miR-31对增殖与侵袭的影响

【数据集背景】

本数据集源站标题为《Differentially Expressed miRNAs in Ewing Sarcoma Compared to Mesenchymal Stem Cells: Low miR-31 Expression with Effects on Proliferation and Invasion》。配套论文《Differentially Expressed miRNAs in Ewing Sarcoma Compared to Mesenchymal Stem Cells: Low miR-31 Expression with Effects on Proliferation and Invasion》发表于 PLoS ONE（2014），DOI：10.1371/journal.pone.0093067。源站首次发布：2014-03-25。

【数据内容】

源站原始描述：Ewing sarcoma, the second most common bone tumor in children and young adults, is an aggressive malignancy with a strong potential to metastasize. Ewing sarcoma is characterised by translocations encoding fusion transcription factors with an EWSR1 transactivation domain fused to an ETS family DNA binding domain. microRNAs are post-transcriptional regulators of gene expression and aberrantly expressed microRNAs have been identified as tumor suppressors or oncogenes in most cancer types. To identi…

【数据结构与技术规格】

文件数 12 个，合计 7.96 MB。Figure_S1.tif：image/tiff，311.68 KB。Figure_S2.tif：image/tiff，1.09 MB。Figure_S3.tif：image/tiff，646.89 KB。数据集 DOI：10.1371/journal.pone.0093067。

【主题与分类】

源站学科分类：Biological Sciences。主题标签：Biochemistry、rna、RNA stability、Nucleic acids、cell biology、Chromosome biology、chromosomes、Chromosome structure and function、Molecular cell biology、Computational biology。

【适用方向】

源站学科分类标注为 Biological Sciences。

【获取与许可】

源站页面：https://plos.figshare.com/articles/dataset/_Differentially_Expressed_miRNAs_in_Ewing_Sarcoma_Compared_to_Mesenchymal_Stem_Cells_Low_miR_31_Expression_with_Effects_on_Proliferation_and_Invasion_/974334

使用许可：CC BY 4.0（https://creativecommons.org/licenses/by/4.0/）。

规范引用：Karnuth, Bianca; Dedy, Nicolas; Spieker, Tilmann; R. Lawlor, Elizabeth; Gattenlöhner, Stefan; Ranft, Andreas; et al. (2016). Differentially Expressed miRNAs in Ewing Sarcoma Compared to Mesenchymal Stem Cells: Low miR-31 Expression with Effects on Proliferation and Invasion. PLOS ONE. Dataset

【使用注意】

· 源站最后修改于 2023-06-03，引用时请注意版本时效。